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Furthermore, neutrophils are able to cause lipid peroxidation in glioblastoma cells by incorporating myeloperoxidase (61)

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At neutral to alkaline pH (above 7), deamidation proceeds through a cyclic imide intermediate that produces a mixture of normal and isomerized products

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IL-4 promotes asymmetric dimethylarginine accumulation, oxo-nitrative stress, and hypoxic response-induced mitochondrial loss in airway epithelial cells